Khoom
Khoom npe: semaglowside
Mechanism ntawm Kev Ua
Semaglutide Simulates lub luag haujlwm ntawm ntuj GLP-1:
1.Qhov insulin zais cia: txhawb cov insulin tso tawm hauv cov ntshav qab zib (ntshav}.
2. Siv menyuam ua kom cov zais glucages: txo cov glycogenolysis hauv daim siab.
3.Delay pais plab khoob: nce satiety thiab txo cov zaub mov noj.
4.Cenal appetite supression: Ua rau cov hypothalamus los txo qhov kev tshaib plab.
Cov khoom lag luam zoo
1.Powerfulfulfulferffer hypoglycemic nyhuv: tseem ceeb txo HBA1C (nce txog 1.5% -2.0%).
2. Kev tswj hwm kev tswj hwm: Qhov hnyav nruab nrab poob ntawm 10% -15% (cov ntaub ntawv kuaj mob).
3.Cardiovascular pab: txo qhov kev pheej hmoo ntawm cov xwm txheej tseem ceeb ntawm myocardicular xwm txheej thiab mob stroke) hauv cov neeg mob muaj hom 2 ntshav qab zib.
Cim npe nrov: high - Qhov hnyav hnyav peptide 20mg semaglutide, Tuam Tshoj siab zoo} zoo} zoo yuag peptide chaw tsim tshuaj paus, lwm tus neeg, Hoobkas